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Hemochromatosis, Cooley Dickinson Hospital Editor: George R Bowers MD CDH Oncology January 2000 Volume 4: No. 1. Hemochromatosis is a common, genetically transmitted disease. The diagnosis of hemochromatosis is sometimes difficult and frequently missed. This issue will review the pathogenesis, diagnosis and treatment of this fascinating disease. Ms. Kathy Fleming will review the management of a most troubling complication of therapy---lymphedema.
Hemochromatosis: diagnosis and management. Bacon BR. Gastroenterology 2001 Feb HFE mutation analysis has strengthened our ability to diagnose HH accurately and is useful in family studies. HFE mutations may play a contributory role in some patients with PCT, NASH, or chronic HCV.
Hemochromatosis, Front Range Gastroenterology Associates, P.C.
2030 Mountain View Avenue, Suite 300, Longmont, CO. Basic statistics
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Hepcidin: A putative iron-regulatory hormone relevant to hereditary hemochromatosis and the anemia of chronic disease. Proc. Natl. Acad. Sci. USA, Vol. 98, Issue 15, 8160-8162, July 17, 2001 The paper by Nicolas et al. in this issue of PNAS (3) presents the exciting possibility that a central player in the communication of body iron stores to the intestinal absorptive cells may have been identified.
Hereditary hemochromatosis. Ann Clin Lab Sci. 1998 Sep-Oct Patients at risk for genetic hemochromatosis should be screened, identified, and treated as early as age 20 to prevent or minimize the deadly complications of hemochromatosis. Population screening should include measurements of serum iron concentration, total iron binding capacity (TIBC), percent saturation of transferrin, and serum ferritin concentrations. Early diagnosis and treatment will reduce the population of aging individuals with severe, complicated hemochromatosis and dramatically reduce medical costs (billions of U.S. dollars per annum) associated with the management of this disease.
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